Human CXCL14 activation kit by CRISPRa

SKU
GA106390
CXCL14 CRISPRa kit - CRISPR gene activation of human C-X-C motif chemokine ligand 14
  $1,657.00
4 Weeks*
Specifications
Specifications
Product Data
Format 3 gRNAs (5ug each), 1 scramble ctrl (10ug) and 1 enhancer vector (10ug)
Target Symbol CXCL14
Locus ID 9547
Components

GA106390G1, CXCL14 gRNA vector 1 in pCas-Guide-GFP-CRISPRa

GA106390G2, CXCL14 gRNA vector 2 in pCas-Guide-GFP-CRISPRa

GA106390G3, CXCL14 gRNA vector 3 in pCas-Guide-GFP-CRISPRa

1 CRISPRa-Enhancer vector, SKU GE100056

1 CRISPRa scramble vector, SKU GE100077

OTI Disclaimer These products are manufactured and supplied by OriGene under license from ERS. The kit is designed based on the best knowledge of CRISPRa SAM technology. The efficiency of the activation can be affected by many factors, including nucleosome occupancy status, chromatin structure and the gene expression level of the target, etc.
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Reference Data
RefSeq NM_004887
UniProt ID O95715
Synonyms BMAC; BRAK; KEC; KS1; MIP-2g; MIP2G; NJAC; SCYB14
Summary This antimicrobial gene belongs to the cytokine gene family which encode secreted proteins involved in immunoregulatory and inflammatory processes. The protein encoded by this gene is structurally related to the CXC (Cys-X-Cys) subfamily of cytokines. Members of this subfamily are characterized by two cysteines separated by a single amino acid. This cytokine displays chemotactic activity for monocytes but not for lymphocytes, dendritic cells, neutrophils or macrophages. It has been implicated that this cytokine is involved in the homeostasis of monocyte-derived macrophages rather than in inflammation. provided by RefSeq, Sep 2014
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Other Versions
SKU Description Size Price
KN402533 CXCL14 - KN2.0, Human gene knockout kit via CRISPR, non-homology mediated. 1 kit
$1,657.00

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*Delivery time may vary from web posted schedule. Occasional delays may occur due to unforeseen complexities in the preparation of your product. International customers may expect an additional 1-2 weeks in shipping.

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