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Celf3 Mouse shRNA Plasmid (Locus ID 78784)

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SKU
TL512056
Celf3 - Mouse, 4 unique 29mer shRNA constructs in lentiviral GFP vector, 5µg of each construct provided
  $1,013.00
2 Weeks*

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Specifications
Specifications
Product Data
Locus ID 78784
Synonyms 4930415M08Rik; BRUNOL1; CAGH4; ERDA4; Tnrc4
Vector pGFP-C-shLenti
E. coli Selection Chloramphenicol (34 ug/ml)
Mammalian Cell Selection Puromycin
Format Lentiviral plasmids
Components Celf3 - Mouse, 4 unique 29mer shRNA constructs in lentiviral GFP vector(Gene ID = 78784). 5µg purified plasmid DNA per construct
29-mer scrambled shRNA cassette in pGFP-C-shLenti Vector, TR30021, included for free.
RefSeq BC057553, NM_001289613, NM_001289616, NM_001289620, NM_172434, NR_110353, NM_001357784, NM_001357785, NM_001357786, NM_001357787, NR_151857, NM_172434.1, NM_172434.2, NM_172434.3, NM_001289620.1, NM_001289616.1, NM_001289613.1, BC031740, NM_001289613.2, NM_172434.4
UniProt ID Q8CIN6
Summary RNA-binding protein involved in the regulation of pre-mRNA alternative splicing. Mediates exon inclusion and/or exclusion in pre-mRNA that are subject to tissue-specific and developmentally regulated alternative splicing. Specifically activates exon 5 inclusion of cardiac isoforms of TNNT2 during heart remodeling at the juvenile to adult transition. Activates the splicing of MAPT/Tau exon 10. Binds to muscle-specific splicing enhancer (MSE) intronic sites flanking the alternative exon 5 of TNNT2 pre-mRNA (By similarity).UniProtKB/Swiss-Prot Function
shRNA Design These shRNA constructs were designed against multiple splice variants at this gene locus. To be certain that your variant of interest is targeted, please contact techsupport@origene.com. If you need a special design or shRNA sequence, please utilize our custom shRNA service.
Storage Store at -20°C
Shipping Ambient
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*Delivery time may vary from web posted schedule. Occasional delays may occur due to unforeseen complexities in the preparation of your product. International customers may expect an additional 1-2 weeks in shipping.

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