BORA Human shRNA Plasmid Kit (Locus ID 79866)

SKU
TL307294
BORA - Human, 4 unique 29mer shRNA constructs in lentiviral GFP vector, 5µg of each construct provided
  $1,013.00
2 Weeks*
Specifications
Specifications
Product Data
Locus ID 79866
Synonyms C13orf34
Vector pGFP-C-shLenti
E. coli Selection Chloramphenicol (34 ug/ml)
Mammalian Cell Selection Puromycin
Format Lentiviral plasmids
Components BORA - Human, 4 unique 29mer shRNA constructs in lentiviral GFP vector(Gene ID = 79866). 5µg purified plasmid DNA per construct
29-mer scrambled shRNA cassette in pGFP-C-shLenti Vector, TR30021, included for free.
RefSeq BC025367, NM_001286746, NM_001286747, NM_024808, NM_024808.1, NM_024808.2, NM_024808.3, NM_001286747.1, NM_001286746.1, BC007119, BC044213, BC056876, BM923278, NM_001366664, NM_024808.5
UniProt ID Q6PGQ7
Summary BORA is an activator of the protein kinase Aurora A (AURKA; MIM 603072), which is required for centrosome maturation, spindle assembly, and asymmetric protein localization during mitosis (Hutterer et al., 2006 PubMed 16890155).supplied by OMIM, Mar 2008
shRNA Design These shRNA constructs were designed against multiple splice variants at this gene locus. To be certain that your variant of interest is targeted, please contact techsupport@origene.com. If you need a special design or shRNA sequence, please utilize our custom shRNA service.
Storage Store at -20°C
Shipping Ambient
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Documents
Resources

file-alt Citations

Bora downregulation results in radioresistance by promoting repair of double strand breaks
Cairns, Peng, Yee et al
PLoS ONE (2015) 10 (3), e0119208 DOI: 10.1371/journal.pone.0119208
Cited by 8
AI Summary: OriGene's Bora shRNA was used to downregulate Bora expression, enabling the researchers to investigate its role in DNA damage repair and radioresistance.
Snippet: CO). Transfection of plasmids and siRNAs was performed with Lipofectamine 2000 and lipofectamine RNAiMAX reagents (Invitrogen, Carlsbad, CA), respectively. Bora shRNA was purchased from Origene (Rockville, MD). Bora ... Cite SaveSaving
Showing 1-1 of 1 papers.
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