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RUNX1 Human shRNA Plasmid Kit (Locus ID 861)

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SKU
TG309685
RUNX1 - Human, 4 unique 29mer shRNA constructs in retroviral GFP vector, 5µg of each construct provided
  $883.00
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Specifications
Specifications
Product Data
Locus ID 861
Synonyms AML1; AML1-EVI-1; AMLCR1; CBF2alpha; CBFA2; EVI-1; PEBP2aB; PEBP2alpha
Vector pGFP-V-RS
E. coli Selection Kanamycin
Mammalian Cell Selection Puromycin
Format Retroviral plasmids
Components RUNX1 - Human, 4 unique 29mer shRNA constructs in retroviral GFP vector(Gene ID = 861). 5µg purified plasmid DNA per construct
29-mer scrambled shRNA cassette in pGFP-V-RS Vector, TR30013, included for free.
RefSeq NM_001001890, NM_001122607, NM_001754, NM_001754.1, NM_001754.2, NM_001754.3, NM_001754.4, NM_001001890.1, NM_001001890.2, NM_001122607.1, BC050363, BC110828, BC136380, BC136381, BC144053, BM149149, NM_001122607.2, NM_001754.5, NM_001001890.3
UniProt ID Q01196
Summary Core binding factor (CBF) is a heterodimeric transcription factor that binds to the core element of many enhancers and promoters. The protein encoded by this gene represents the alpha subunit of CBF and is thought to be involved in the development of normal hematopoiesis. Chromosomal translocations involving this gene are well-documented and have been associated with several types of leukemia. Three transcript variants encoding different isoforms have been found for this gene. provided by RefSeq, Jul 2008
shRNA Design These shRNA constructs were designed against multiple splice variants at this gene locus. To be certain that your variant of interest is targeted, please contact techsupport@origene.com. If you need a special design or shRNA sequence, please utilize our custom shRNA service.
Storage Store at -20°C
Shipping Ambient
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Documents
Resources

file-alt Citations

MiR-18a increased the permeability of BTB via RUNX1 mediated down-regulation of ZO-1, occludin and claudin-5
Miao, Zhao, Zhao et al
Cell. Signal. (2014) 27 (1), 156-167 DOI: 10.1016/j.cellsig.2014.10.008
Cited by 55
AI Summary: In this study, OriGene's pCMV6-XL5 vector and pGFP-V-RS vector were used to overexpress and silence RUNX1, respectively, enabling the authors to investigate the role of RUNX1 in miR-18a-mediated regulation of blood-tumor barrier permeability.
Snippet: To further verify whether RUNX1 involves in the regulation of miR-18a on the permeability of BTB, the overexpression and silence of RUNX1 were respectively performed with pCMV6-XL5 vector and pGFP-V-RS vector (Origene, Rockville MD, USA). ... Cite SaveSaving
Showing 1-1 of 1 papers.
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