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Cox5b (NM_009942) Mouse Recombinant Protein

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SKU
TP500704
Purified recombinant protein of Mouse cytochrome c oxidase subunit 5B (Cox5b), with C-terminal MYC/DDK tag, expressed in HEK293T cells, 20ug
Size:
20 ug
  $1,020.00
4 Weeks*
Bulk/Customize

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Specifications
Specifications
Product Data
Species Mouse
Expression Host HEK293
Expression cDNA Clone or AA Sequence
Protein Sequence
A DNA sequence from Mouse cDNA ORF Clone, MR200704, encoding Mouse full-length Cox5b.
Tag C-MYC/DDK
Predicted MW 14.3 kDa
Concentration >0.05 µg/µL as determined by microplate BCA method
Purity > 80% as determined by SDS-PAGE and Coomassie blue staining
Buffer 25 mM Tris-HCl, 100 mM glycine, pH 7.3, 10% glycerol
Note For testing in cell culture applications, please filter before use. Note that you may experience some loss of protein during the filtration process.
Storage Store at -80°C after receiving vials.
Stability Stable for 12 months from the date of receipt of the product under proper storage and handling conditions. Avoid repeated freeze-thaw cycles.
Shipping Dry Ice
Reference Data
RefSeq NP_034072
Locus ID 12859
UniProt ID Q9D881
Cytogenetics 1 B
RefSeq Size 584
RefSeq ORF 387
Synonyms MGC117683
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Documents
Resources

file-alt Citations

COX5B-Mediated Bioenergetic Alterations Modulate Cell Growth and Anticancer Drug Susceptibility by Orchestrating Claudin-2 Expression in Colorectal Cancers
Chu, Lim, Yeh et al
Biomedicines (2021) 10 (1) DOI: 10.3390/biomedicines10010060
Cited by 18
AI Summary: In this study, OriGene’s COX5B shRNA (cat. no. TG313691) and CLDN2 cDNA (cat. no. RC210794) products were used to manipulate the expression of COX5B and Claudin-2, enabling the authors to investigate their roles in regulating cell growth and drug susceptibility in colorectal cancer cells.
FigureFigureFigure
Showing 1-1 of 1 papers.
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*Delivery time may vary from web posted schedule. Occasional delays may occur due to unforeseen complexities in the preparation of your product. International customers may expect an additional 1-2 weeks in shipping.

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