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Candida albicans (Wild type) Human Protein

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SKU
AR00054PU-N
Candida albicans (Wild type) protein, 1 mg
Size:
1 mg
  $1,785.00
2 Weeks*
Bulk/Customize
Specifications
Specifications
Product Data
Species Human
Protein Source Cell culture
Concentration lot specific
Purity Washed harvested organisms are extracted during a proprietary process. The resulting antigen consists of
cytoplasmic components and some cellular material.
Presentation State Purified
Buffer Presentation State: Purified
State: Liquid purified purified protein
Buffer System: Glycine buffer without preservatives
Preparation Liquid purified purified protein
Recommended Dilution Suitable for use in ELISA.
Protein Description Candida albicans (Wild Type) Antigen
Note Centrifuge before opening to ensure complete recovery of vial contents.

Caution: No test guarantees a product to be non-infectious. All materials should be handled as if potentially infectious. Generally accepted laboratory practices appropriate for infectious materials should be employed when handling this product.
Storage Store the protein at -20°C to -80°C.
Avoid repeated freezing and thawing.
Stability Shelf life: one year from despatch.
Shipping Blue Ice
Reference Data
Synonyms C. albicans
Summary Candida albicans is the most frequently isolated fungal pathogen of humans, affecting immunocompromised patients ranging from premature infants to AIDS sufferers. Systemic infections have an attributed mortality of 30-50%. C. albicans is a diploid organism which has eight sets of homologous chromosomes. It has a genome of approximately 16 Mb (haploid), about 30% greater than S. cerevisiae (baker’s yeast).
Protein Families Suitable for use in ELISA.
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Documents
Resources

file-alt Citations

Impaired SorLA maturation and trafficking as a new mechanism for SORL1 missense variants in Alzheimer disease
Rovelet-Lecrux, Feuillette, Miguel et al
Acta Neuropathol Commun (2021) 9 (1), 196 DOI: 10.1186/s40478-021-01294-4
Cited by 40
AI Summary: OriGene’s pCMV6-XL5-WT-SorLAFL plasmid was used to overexpress wild-type SorLA in cell models, enabling the functional screening and analysis of SORL1 missense variants’ effects on SorLA maturation and trafficking in this Alzheimer’s disease study.
Snippet: (pCMV6-XL5-WT-SorLAFL OriGene Technologies, Inc, Rockville, MD, USA
FigureFigureFigure
Alterations in Protein Kinase A Substrate Specificity as a Potential Cause of Cushing Syndrome
Bathon, Weigand, Vanselow et al
Endocrinology (2019) 160 (2), 447-459 DOI: 10.1210/en.2018-00775
AI Summary: OriGene contributed to this study by providing the plasmid encoding human wild-type Cα, which was used as a template to generate PRKACA mutants for investigating alterations in protein kinase A substrate specificity related to Cushing syndrome.
Snippet: Methods Plasmids Plasmids encoding human RIα, RIIβ and RIIβ-FLAG were described earlier (13). A plasmid encoding human wild-type Cα was purchased from Origene. Mutations in the coding sequence of Cα were introduced by PCR as previously described (10
Mineralocorticoid Receptor (MR) Trans-Activation of Inflammatory AP-1 Signaling: Dependence on DNA Sequence, MR Conformation, and AP-1 Family Member Expression
Dougherty, Elinoff, Ferreyra et al
J. Biol. Chem. (2016)
AI Summary: OriGene products, specifically the DDK-myc/pCMV6 empty vector, human GR-DDK-myc/pCMV6, and MR-DDK-myc/pCMV6 plasmids, were used to transiently transfect HEK293 cells for overexpression studies of glucocorticoid and mineralocorticoid receptors, enabling investigation of their roles in inflammatory AP-1 signaling.
Snippet: Forty-eight hours prior to transfection, 5x105 HEK293 cells were seeded onto 35 mm plates. Cells were transiently transfected with 1 µg of empty-vector (DDK-myc/pCMV6;Origene, Rockville, MD) plus 0.5 µg of human GRDDK-myc/pCMV6 (Origene), MR-DDKmyc/pCMV6 (Origene), or additional empty-vector using lipofectamine 2000 (Life Technologies)
Neuronal-specific overexpression of a mutant valosin-containing protein associated with IBMPFD promotes aberrant ubiquitin and TDP-43 accumulation and cognitive dysfunction in transgenic mice
Rodriguez-Ortiz, Hoshino, Cheng et al
Am. J. Pathol. (2013) 183 (2), 504-15 DOI: 10.1016/j.ajpath.2013.04.014
Cited by 34
AI Summary: OriGene’s human wild-type VCP product was used to generate transgenic mice in this study, enabling investigation of how mutant VCP contributes to neurodegeneration and cognitive dysfunction.
Snippet: Generation of Tg Mice. Human wild-type VCP was purchased from OriGene Technologies Inc. ... For PCR genotyping, forward primer 5′-GAGGTATTCATCATGTGCT-3′ and reverse primer 5′-AAGGACGATGCAAACAGCTT-3′ liberated a 500-bp band from Tg mice.
Gαi2 signaling promotes skeletal muscle hypertrophy, myoblast differentiation, and muscle regeneration
Minetti, Feige, Rosenstiel et al
Sci Signal (2011) 4 (201), ra80 DOI: 10.1126/scisignal.2002038
Cited by 86
AI Summary: OriGene contributed to this study by providing the expression vector encoding wild-type human PKCα, which was used to investigate the role of protein kinase C in skeletal muscle hypertrophy and differentiation.
Snippet: obtained from UMR cDNA Resource Center, University of Missouri–Rolla. The expression vector encoding wild-type human PKCα was purchased from Origene. The constitutively active human PKCα (PKCα ca) contains an
Showing 1-5 of 5 papers.
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