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CXCR4 Human shRNA Plasmid Kit (Locus ID 7852)

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SKU
TR313630
CXCR4 - Human, 4 unique 29mer shRNA constructs in retroviral untagged vector, 5µg of each construct provided
  $883.00
In Stock*

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Specifications
Specifications
Product Data
Locus ID 7852
Synonyms CD184; D2S201E; FB22; HM89; HSY3RR; LAP-3; LAP3; LCR1; LESTR; NPY3R; NPYR; NPYRL; NPYY3R; WHIM; WHIMS
Vector pRS
E. coli Selection Ampicillin
Mammalian Cell Selection Puromycin
Format Retroviral plasmids
Components CXCR4 - Human, 4 unique 29mer shRNA constructs in retroviral untagged vector(Gene ID = 7852). 5µg purified plasmid DNA per construct
29-mer scrambled shRNA cassette in pRS Vector, TR30012, included for free.
RefSeq NM_001008540, NM_003467, NM_001348056, NM_001348059, NM_001348060, NM_003467.1, NM_003467.2, NM_001008540.1, NM_001008540.2, BC020968, BC020968.2, NM_003467.3
UniProt ID P61073
Summary This gene encodes a CXC chemokine receptor specific for stromal cell-derived factor-1. The protein has 7 transmembrane regions and is located on the cell surface. It acts with the CD4 protein to support HIV entry into cells and is also highly expressed in breast cancer cells. Mutations in this gene have been associated with WHIM (warts, hypogammaglobulinemia, infections, and myelokathexis) syndrome. Alternate transcriptional splice variants, encoding different isoforms, have been characterized. provided by RefSeq, Jul 2008
shRNA Design These shRNA constructs were designed against multiple splice variants at this gene locus. To be certain that your variant of interest is targeted, please contact techsupport@origene.com. If you need a special design or shRNA sequence, please utilize our custom shRNA service.
Storage Store at -20°C
Shipping Ambient
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Documents
Resources

file-alt Citations

EZH2-mediated loss of miR-622 determines CXCR4 activation in hepatocellular carcinoma
Liu, Liu, Liu et al
Nat Commun (2015) 6, 8494 DOI: 10.1038/ncomms9494
Cited by 137
AI Summary: In this study, OriGene products including EZH2 shRNA (OriGene, #TG311690) and CXCR4 shRNA (OriGene, #TG312699) were used to knock down EZH2 and CXCR4 expression, respectively, enabling the researchers to investigate the molecular mechanisms regulating CXCR4 in hepatocellular carcinoma.
Snippet: Cite SaveSaving
Showing 1-1 of 1 papers.
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