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FANCA Human shRNA Plasmid Kit (Locus ID 2175)

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SKU
TR313071
FANCA - Human, 4 unique 29mer shRNA constructs in retroviral untagged vector, 5µg of each construct provided
  $883.00
In Stock*

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Specifications
Specifications
Product Data
Locus ID 2175
Synonyms FA; FA-H; FA1; FAA; FACA; FAH; FANCH
Vector pRS
E. coli Selection Ampicillin
Mammalian Cell Selection Puromycin
Format Retroviral plasmids
Components FANCA - Human, 4 unique 29mer shRNA constructs in retroviral untagged vector(Gene ID = 2175). 5µg purified plasmid DNA per construct
29-mer scrambled shRNA cassette in pRS Vector, TR30012, included for free.
RefSeq NM_000135, NM_001018112, NM_001286167, NM_001351830, NM_000135.1, NM_000135.2, NM_001018112.1, NM_001286167.1, BC008979, BC022498, BC064540, BC120978, BC120979, BC127633, BC141971, BC141972, BC156522, BC172515, NM_000135.4, NM_001286167.3, NM_001018112.3
UniProt ID O15360
Summary The Fanconi anemia complementation group (FANC) currently includes FANCA, FANCB, FANCC, FANCD1 (also called BRCA2), FANCD2, FANCE, FANCF, FANCG, FANCI, FANCJ (also called BRIP1), FANCL, FANCM and FANCN (also called PALB2). The previously defined group FANCH is the same as FANCA. Fanconi anemia is a genetically heterogeneous recessive disorder characterized by cytogenetic instability, hypersensitivity to DNA crosslinking agents, increased chromosomal breakage, and defective DNA repair. The members of the Fanconi anemia complementation group do not share sequence similarity; they are related by their assembly into a common nuclear protein complex. This gene encodes the protein for complementation group A. Alternative splicing results in multiple transcript variants encoding different isoforms. Mutations in this gene are the most common cause of Fanconi anemia. provided by RefSeq, Jul 2008
shRNA Design These shRNA constructs were designed against multiple splice variants at this gene locus. To be certain that your variant of interest is targeted, please contact techsupport@origene.com. If you need a special design or shRNA sequence, please utilize our custom shRNA service.
Storage Store at -20°C
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Documents
Resources

file-alt Citations

Upregulated ATM gene expression and activated DNA crosslink-induced damage response checkpoint in Fanconi anemia: implications for carcinogenesis
Yamamoto, Nihrane, Aglipay et al
Mol Med (2008) 14 (3-4), 167-74 DOI: 10.2119/2007-00122.Yamamoto
AI Summary: OriGene products contributed to this study by providing the human FANCA cDNA clone (OriGene, SC117675), which was used for functional analysis of FANCA in primary fibroblasts to investigate DNA damage response mechanisms in Fanconi anemia.
Showing 1-2 of 2 papers.
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