CLEC2D (NM_001004419) Human Tagged ORF Clone Lentiviral Particle

SKU
RC221680L1V
Lenti ORF particles, CLEC2D (Myc-DDK tagged) - Human C-type lectin domain family 2, member D (CLEC2D), transcript variant 2, 200ul, >10^7 TU/mL
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    LentiORF®

    Expression-ready ORF plasmid in lenti backbone

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    Ready-to-use Lentiviral Particles

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$850.00
3 Weeks*
Specifications
Product Data
Type Human Tagged ORF Clone Lentiviral Particle
Tag Myc-DDK
Target Symbol CLEC2D
Synonyms CLAX; LLT1; OCIL
Vector pLenti-C-Myc-DDK
Mammalian Cell Selection None
Sequence Data
ORF Nucleotide Sequence
The ORF insert of this clone is exactly the same as(RC221680).
ACCN NM_001004419
ORF Size 582 bp
OTI Disclaimer The molecular sequence of this clone aligns with the gene accession number as a point of reference only. However, individual transcript sequences of the same gene can differ through naturally occurring variations (e.g. polymorphisms), each with its own valid existence. This clone is substantially in agreement with the reference, but a complete review of all prevailing variants is recommended prior to use. More info
OTI Annotation This clone was engineered to express the complete ORF with an expression tag. Expression varies depending on the nature of the gene.
Shipping Dry Ice
Reference Data
RefSeq NM_001004419.1
RefSeq Size 1821 bp
RefSeq ORF 585 bp
Locus ID 29121
UniProt ID Q9UHP7
Cytogenetics 12p13.31
Protein Families Druggable Genome, Transmembrane
MW 22.1 kDa
Summary This gene encodes a member of the natural killer cell receptor C-type lectin family. The encoded protein inhibits osteoclast formation and contains a transmembrane domain near the N-terminus as well as the C-type lectin-like extracellular domain. Several alternatively spliced transcript variants have been identified for this gene. [provided by RefSeq, Oct 2010]
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*Delivery time may vary from web posted schedule. Occasional delays may occur due to unforeseen complexities in the preparation of your product. International customers may expect an additional 1-2 weeks in shipping.