RALBP1 (NM_006788) Human Tagged ORF Clone Lentiviral Particle

SKU
RC201524L2V
Lenti ORF particles, RALBP1 (mGFP-tagged) - Human ralA binding protein 1 (RALBP1), 200ul, >10^7 TU/mL
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    Expression-ready ORF plasmid in lenti backbone

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$1,160.00
5 Weeks*
Specifications
Product Data
Type Human Tagged ORF Clone Lentiviral Particle
Tag mGFP
Target Symbol RALBP1
Synonyms RIP1; RLIP1; RLIP76
Vector pLenti-C-mGFP
Mammalian Cell Selection None
Sequence Data
ORF Nucleotide Sequence
The ORF insert of this clone is exactly the same as(RC201524).
ACCN NM_006788
ORF Size 1965 bp
OTI Disclaimer The molecular sequence of this clone aligns with the gene accession number as a point of reference only. However, individual transcript sequences of the same gene can differ through naturally occurring variations (e.g. polymorphisms), each with its own valid existence. This clone is substantially in agreement with the reference, but a complete review of all prevailing variants is recommended prior to use. More info
OTI Annotation This clone was engineered to express the complete ORF with an expression tag. Expression varies depending on the nature of the gene.
Shipping Dry Ice
Reference Data
RefSeq NM_006788.3
RefSeq Size 4368 bp
RefSeq ORF 1968 bp
Locus ID 10928
UniProt ID Q15311
Cytogenetics 18p11.22
Domains RhoGAP
Protein Pathways Pancreatic cancer, Pathways in cancer
MW 75.9 kDa
Summary RALBP1 plays a role in receptor-mediated endocytosis and is a downstream effector of the small GTP-binding protein RAL (see RALA; MIM 179550). Small G proteins, such as RAL, have GDP-bound inactive and GTP-bound active forms, which shift from the inactive to the active state through the action of RALGDS (MIM 601619), which in turn is activated by RAS (see HRAS; MIM 190020) (summary by Feig, 2003 [PubMed 12888294]).[supplied by OMIM, Nov 2010]
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*Delivery time may vary from web posted schedule. Occasional delays may occur due to unforeseen complexities in the preparation of your product. International customers may expect an additional 1-2 weeks in shipping.