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PER1 (NM_002616) Human Tagged ORF Clone Lentiviral Particle
CAT#: RC224777L4V
- LentiORF®
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Lenti ORF particles, PER1 (mGFP-tagged)-Human period homolog 1 (Drosophila) (PER1), 200ul, >10^7 TU/mL
Lentiviral Particles: DDK DDK w/ Puro mGFP
Buy this product and get 50% off on the Lenti RapidTiter kit. Use Code: Rapid50
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USD 365.00
Specifications
| Product Data | |
| Type | Human Tagged ORF Clone Lentiviral Particle |
| Tag | mGFP |
| Symbol | PER1 |
| Synonyms | hPER; PER; RIGUI |
| Mammalian Cell Selection | Puromycin |
| Vector | pLenti-C-mGFP-P2A-Puro |
| ACCN | NM_002616 |
| ORF Size | 3870 bp |
| Sequence Data |
The ORF insert of this clone is exactly the same as(RC224777).
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| OTI Disclaimer | The molecular sequence of this clone aligns with the gene accession number as a point of reference only. However, individual transcript sequences of the same gene can differ through naturally occurring variations (e.g. polymorphisms), each with its own valid existence. This clone is substantially in agreement with the reference, but a complete review of all prevailing variants is recommended prior to use. More info |
| OTI Annotation | This clone was engineered to express the complete ORF with an expression tag. Expression varies depending on the nature of the gene. |
| Reference Data | |
| RefSeq | NM_002616.1, NP_002607.1 |
| RefSeq Size | 4656 bp |
| RefSeq ORF | 3873 bp |
| Locus ID | 5187 |
| UniProt ID | O15534 |
| Cytogenetics | 17p13.1 |
| Protein Families | Druggable Genome |
| Protein Pathways | Circadian rhythm - mammal |
| MW | 136 kDa |
| Gene Summary | This gene is a member of the Period family of genes and is expressed in a circadian pattern in the suprachiasmatic nucleus, the primary circadian pacemaker in the mammalian brain. Genes in this family encode components of the circadian rhythms of locomotor activity, metabolism, and behavior. This gene is upregulated by CLOCK/ARNTL heterodimers but then represses this upregulation in a feedback loop using PER/CRY heterodimers to interact with CLOCK/ARNTL. Polymorphisms in this gene may increase the risk of getting certain cancers. Alternative splicing has been observed in this gene; however, these variants have not been fully described. [provided by RefSeq, Jan 2014] |
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