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Home Antibody All anti-PEX5 antibodies

Anti-PEX5 TRUEMAB Antibody clone 6E9

TrueMAB™ Antibodies - Made against Authentic Protein Antigens

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Specifications Citations Related Products Product Documents
SKU Description Amount Price Availability*  
TA501430
  • anti-PEX5 mouse monoclonal antibody, clone 6E9
  • Free Sample of Positive Control: HEK293T cell transient overexpression lysate (LC424800) , 20ug Explanation
100ul 379 In Stock
CF501430
  • Carrier-free (BSA/glycerol-free) PEX5 mouse monoclonal antibody, clone 6E9
100ug $450 3-4 weeks
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WB(2)
IF(1)
FC(1)
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Also for PEX5 (NM_000319)
cDNA Clone shRNA/siRNA Lysate Protein Antibody

OriGene Data

ImmunogenFull length human recombinant protein of human PEX5 (NP_000310) produced in HEK293T cell.
Clone Nameclone 6E9 IsotypeIgG1
Species ReactivityHuman, Dog, Rat, Monkey Concentration0.57 mg/ml
Guaranteed Application *WB, IF, FC Suggested DilutionsWB 1:500~2000, IF 1:100, FLOW 1:100
BufferPBS (PH 7.3) containing 1% BSA, 50% glycerol and 0.02% sodium azide.
Purification Purified from mouse ascites fluids by affinity chromatography

Reference Data

Target NameHomo sapiens peroxisomal biogenesis factor 5 (PEX5), transcript variant 2
Alternative NamePBD2A; PBD2B; PTS1-BP; PTS1R; PXR1
Database LinkNP_000310
Entrez Gene 5830 Human
Entrez Gene 312703 Rat
Entrez Gene 715173 Monkey
Entrez Gene 486710 Dog
FunctionThe product of this gene binds to the C-terminal PTS1-type tripeptide peroxisomal targeting signal (SKL-type) and plays an essential role in peroxisomal protein import. Peroxins (PEXs) are proteins that are essential for the assembly of functional peroxisomes. The peroxisome biogenesis disorders (PBDs) are a group of genetically heterogeneous autosomal recessive, lethal diseases characterized by multiple defects in peroxisome function. The peroxisomal biogenesis disorders are a heterogeneous group with at least 14 complementation groups and with more than 1 phenotype being observed in cases falling into particular complementation groups. Although the clinical features of PBD patients vary, cells from all PBD patients exhibit a defect in the import of one or more classes of peroxisomal matrix proteins into the organelle. Defects in this gene are a cause of neonatal adrenoleukodystrophy (NALD), a cause of Zellweger syndrome (ZWS) as well as may be a cause of infantile Refsum disease (IRD). Alternatively spliced transcript variants encoding different isoforms have been identified. [provided by RefSeq].
Related Pathway

* Availability is in business days
* OriGene provides validated application data and protocol, with money back guarantee.

WB Image
HEK293T cells were transfected with the pCMV6-ENTRY control (Left lane) or pCMV6-ENTRY PEX5 (RC202062, Right lane) cDNA for 48 hrs and lysed. Equivalent amounts of cell lysates (5 ug per lane) were separated by SDS-PAGE and immunoblotted with anti-PEX5.
WB Image
Western blot analysis of extracts (35ug) from 9 different cell lines by using anti-PEX5 monoclonal antibody.
IF Image
Anti-PEX5 mouse monoclonal antibody (TA501430) immunofluorescent staining of COS7 cells transiently transfected by pCMV6-ENTRY PEX5(RC202062).
FC Image
HEK293T cells transfected with either RC202062 overexpress plasmid(Red) or empty vector control plasmid(Blue) were immunostained by anti-PEX5 antibody(TA501430), and then analyzed by flow cytometry.

 

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