CD36 Mouse Monoclonal Antibody [Clone ID: OTI3F4]

CAT#: TA400017

Anti-CD36 mouse mAb, clone OTI3F4, HRP conjugated

Product Images

HEK293T cells were transfected with pCMV6-ENTRY control (Lane 1) or pCMV6-ENTRY CD36 (Lane 2) plasmids for 48 hrs. Equivalent amounts of cell lysates (5 ug per lane) were separated by SDS-PAGE and immunoblotted with anti-CD36- HRP.

Specifications

Product Data

• Clone Name: OTI3F4
• Applications: WB
• Recommended Dilution: WB: 1:1000
• Reactivities: Human
• Host: Mouse
• Isotype: IgG1
• Clonality: Monoclonal
• Immunogen: Full length human recombinant protein of human CD36 (NP_000063) produced in HEK293T cell.
• Formulation: PBS (pH7.4) containing 50% glycerol, 0.1% BSA
• Concentration: 0.5 mg/ml
• Purification: Purified from cell culture supernatant by affinity chromatography
• Conjugation: HRP
• Storage: Store at -20°C as received.
• Stability: Stable for 12 months from date of receipt.
• Predicted Protein Size: 53.0 kDa
• Gene Name: CD36 molecule
• Database Link:
  NP_000063
  Entrez Gene 948 Human
  UniProt ID P16671
• Background: The protein encoded by this gene is the fourth major glycoprotein of the platelet surface and serves as a receptor for thrombospondin in platelets and various cell lines. Since thrombospondins are widely distributed proteins involved in a variety of adhesive processes, this protein may have important functions as a cell adhesion molecule. It binds to collagen, thrombospondin, anionic phospholipids and oxidized LDL. It directly mediates cytoadherence of Plasmodium falciparum parasitized erythrocytes and it binds long chain fatty acids and may function in the transport and/or as a regulator of fatty acid transport. Mutations in this gene cause platelet glycoprotein deficiency. Multiple alternatively spliced transcript variants encoding the same protein have been found for this gene.
• Synonyms: BDPLT10; CHDS7; FAT; GP3B; GP4; GPIV; PASIV; SCARB3

Reference Data

• Protein Families: Druggable Genome, Transmembrane
• Protein Pathways: Adipocytokine signaling pathway, ECM-receptor interaction, Hematopoietic cell lineage, PPAR signaling pathway