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Home cDNA Clone TrueORF All CNTN4 ORF Clones

CNTN4 (NM_175612) Human cDNA ORF Clone

Specifications Citations Clones of Other Species Product Documents
Cat. No. Description Price Availability  
RG214500 CNTN4 (GFP-tagged) - Human contactin 4 (CNTN4), transcript variant 2, 10µg
$510
In Stock
TA150041 2H8, Anti-tGFP monoclonal antibody, 100µl $248 In Stock
Clone Modification
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TrueORF Data for RG214500
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Vector: pCMV6-AC-GFP   Change vector? Tag: C-terminal TurboGFP
Sequence Data: ORF Nucleotide Sequence
Protein Sequence
ORF Size: 849 bp
Restriction Sites: SgfI-MluI     Cloning Scheme for this gene     Plasmid Map Plasmid Map
OTI Annotation: This clone was engineered to express the complete ORF with an expression tag.
OTI Disclaimer: The molecular sequence of this clone aligns with the gene accession number as a point of reference only. However, individual transcript sequences of the same gene can differ through naturally occurring variations (e.g. polymorphisms), each with its own valid existence. This clone is substantially in agreement with the reference, but a complete review of all prevailing variants is recommended prior to use. More info
Product Components: The ORF clone is ion-exchange column purified, transfection-ready dried plasmid DNA, and shipped with 2 vector sequencing primers.
Protein Families: Secreted Protein

Reference Data
RefSeq: NM_175612.1, NP_783301
RefSeq Size: 3072 RefSeq ORF: 848
Synonyms : AXCAM; BIG-2; CNTN4A; MGC33615; BIG-2; CNTN4A; MGC33615; OTTHUMP00000147566; OTTHUMP00000147567; axonal cell adhesion molecule; axonal-associated cell adhesion molecule; neural cell adhesion protein BIG-2; contactin 4
LocusID: 152330 Cytogenetic: 3p26.3
Summary: This gene encodes a member of the contactin family of immunoglobulins. Contactins are axon-associated cell adhesion molecules that function in neuronal network formation and plasticity. The encoded protein is a glycosylphosphatidylinositol-anchored neuronal membrane protein that may play a role in the formation of axon connections in the developing nervous system. Deletion or mutation of this gene may play a role in 3p deletion syndrome and autism spectrum disorders. Alternative splicing results in multiple transcript variants. [provided by RefSeq, May 2011].

 

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